Insulin Resistance and Nonalcoholic Fatty Liver Disease (Nafld) / Nonalcoholic Steatohepatitis (Nash)
by Yoneda, MasatoEdition: 1st
Format: Paperback
Pub. Date: 2010-10-01
Publisher(s): Nova Science Pub Inc
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Summary
The liver is the first organ to receive nutrients that enter the body via the intestines after a meal, and this organ plays a pivotal role in energy metabolism. Non-alcoholic fatty liver disease (NAFLD) is among the most common causes of chronic liver disease in the world and is now considered to be a component of metabolic syndrome. A wide spectrum of histological changes has been observed in NAFLD, ranging from simple steatosis, which is generally non-progressive, to nonalcoholic steatohepatitis (NASH), liver cirrhosis, liver failure, and sometimes even hepatocellular carcinoma. This book reviews recent evidence concerning insulin resistance and NAFLD/NASH as a step towards a comprehensive understanding of the pathophysiology and treatment of NAFLD/NASH.
Table of Contents
| Introduction | |
| NAFLD & Insulin Resistance (IR) | |
| Pathophysiology of NASH: Insulin Resistance, Free Fatty Acids & Cytokines | |
| Adiponectin & NASH | |
| TNF-a & NAFLD/NASH a | |
| Leptin & NAFLD/NASH | |
| Peroxisome Proliferators-Activated Receptors (PPARs) & NAFLD/NASH | |
| Iron Overload & NAFLD/NASH | |
| Mitochondrial Abnormalities & NAFLD/NASH | |
| Genetic Influences in NAFLD / NASH | |
| Therapeutic Strategy for NASH Patients | |
| Natural History of NAFLD | |
| Conclusions | |
| Index | |
| Table of Contents provided by Publisher. All Rights Reserved. |
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